Non-convulsive epiletic seizure after electroconvulsive therapy session

Introduction Electroconvulsive therapy (ECT) is a procedure performed under general anaesthesia involving triggering an intentional brief seizure through small electrical currents the brain. The anaesthetic depth should be adequate prior to shock and measured with BIS, processed electroencephalogram (EEG) monitor. Adjusting hypnotic dose allows decrease ictal threshold thus improve response treatment side effects. Objectives Our goal detecting elements such as spontaneous epileptiform activity after ECT without tonic-clonic spectral density matrix (SDM). Methods patient: 87-year-old woman, diagnosed F20.2 catatonic schizophrenia antipsychotic since her youth. She has required multiple hospital admissions due psychopathological decompensations until starting monthly maintenance sessions in 2014. Since then she no new have been required. Results Images 1D 1E shows SDM, spectrogram of EEG. X axis show time (minutes), Y frequency (Hz) Z energy or intensity that bands replaced by colors: warm colors (red) reflecting high electroencephalographic cool (yellow, blue green), low activity. 2 3’s EDM electroconvulsive We observed initial awake patient, colours practically all bands, including beta band (13-30 Hz), characteristic waking states. Around 9:50 induction occurs, increases slow frequencies (red alpha, theta delta), plus increase cold beta, disappearance brain frequency. asterisk reflects EEG discharge, followed postcritical state exclusively waves amplitude (delta some theta) absence other (blue colour alpha bands) around 9:57. At about 10:00 there abrupt appearance (warm colours) delta, mainly, clinically reflected patient absent disconnected from environment, but New which predominates, little activity, warmer reappear progressive recovery wakefulness. Image: Image 2: 3: Conclusions Psychiatric pathology can observe changes EEG, correcting stimulus anesthetic appropriate trigger anesthesia. Disclosure Interest None Declared